I can’t write about ‘viruses’ and ‘antibodies’ without feeling like Alice in Wonderland, or Dorothy on the Yellow Brick Road ... this road of virology and immunology as we know it today is a muck-filled trench of contradictions and non-sense. Here’s a sentence from a presentation in 2020 on Lyme that sounds very much like what we know about the computer-generated SARS ‘virus’. “The strain used for lab testing (B31) was a lab strain and never collected from a real human patient.”
ANTI-BODIES (think about that word for a minute) .... SCIENCE OF PROTEINS https://viroliegy.com/category/antibodies/
In Summary:
• Antibodies are said to provide potent protection against the flu and other “viruses”
• Vaccines are designed to stimulate antibody production
• Researchers measure antibodies to gauge the strength of the immune response to the infection
• However, questions remain about immune factors that drive the magnitude of the antibody response, including why flu infections do not always trigger a rise in blood antibodies
• The immune factors that explain why some people mount a strong antibody response and others don’t has not been well understood
• Airway monocytes often show up as correlates of severe disease, yet they were associated with protective immune responses in a recent study on influenza
• This was said to point to the “fact” that many immune factors play protective and pathogenic roles at the same time
• Antibodies are said to provide potent protection against the flu and other “viruses”
• Vaccines are designed to stimulate antibody production
• Researchers measure antibodies to gauge the strength of the immune response to the infection
• However, questions remain about immune factors that drive the magnitude of the antibody response, including why flu infections do not always trigger a rise in blood antibodies
• The immune factors that explain why some people mount a strong antibody response and others don’t has not been well understood
• Airway monocytes often show up as correlates of severe disease, yet they were associated with protective immune responses in a recent study on influenza
• This was said to point to the “fact” that many immune factors play protective and pathogenic roles at the same time
“Taken all together, these studies provide indirect evidence that the unseen hypothetical/theoretical particles known as antibodies can be found in some cases of disease. These studies also provide evidence that these unseen hypothetical/theoretical particles are not found in many cases of disease. Antibodies may be associated with more severe disease but are also not present in cases of severe disease. They may be present in asymptomatic/mild cases of disease but are also not present in many asymptomatic/mild cases of disease.
The HAI gold standard of antibody titers > 40 has been correlated with “protection” in a few studies but this level has also been shown to be completely ineffective at providing “protection” in many studies post vaccination.
The problem for immunology and “antibodies” is that, just as in the case with virology and “viruses,” they rely primarily on correlation equaling causation. However, these correlations are inconsistent at best and many times non-existent and/or contradicted in future studies. Instead of admitting that they may be wrong by throwing out the old theories and starting over, the researchers try to cram new, contradictory findings/theories into the existing theory thus creating a nearly incomprehensible and overly confusing mosaic made up of pieces from completely unrelated puzzles.
This is why they can claim antibodies are “protective” even though they are not found in many cases of disease. This is how they can state that antibodies are a sign the immune system is working in regards to vaccinations yet they are also a sign one has a weakened immune system with HIV. This is why the presence of antibodies mean one has successfully fought off a disease and will be “protected” from future infections yet in the case of Dengue Fever, antibodies will make a reinfection worse and potentially fatal.
This is how we end up with multiple immune systems with factors that can be simultaneously protective and pathogenic. It is a long chain of confusing, confounding, and contradictory indirect evidence all rolled up into evolving incomprehensible theories. In other words, it’s a mess.” ~ Mike Stone, viroLIEgy.com
The HAI gold standard of antibody titers > 40 has been correlated with “protection” in a few studies but this level has also been shown to be completely ineffective at providing “protection” in many studies post vaccination.
The problem for immunology and “antibodies” is that, just as in the case with virology and “viruses,” they rely primarily on correlation equaling causation. However, these correlations are inconsistent at best and many times non-existent and/or contradicted in future studies. Instead of admitting that they may be wrong by throwing out the old theories and starting over, the researchers try to cram new, contradictory findings/theories into the existing theory thus creating a nearly incomprehensible and overly confusing mosaic made up of pieces from completely unrelated puzzles.
This is why they can claim antibodies are “protective” even though they are not found in many cases of disease. This is how they can state that antibodies are a sign the immune system is working in regards to vaccinations yet they are also a sign one has a weakened immune system with HIV. This is why the presence of antibodies mean one has successfully fought off a disease and will be “protected” from future infections yet in the case of Dengue Fever, antibodies will make a reinfection worse and potentially fatal.
This is how we end up with multiple immune systems with factors that can be simultaneously protective and pathogenic. It is a long chain of confusing, confounding, and contradictory indirect evidence all rolled up into evolving incomprehensible theories. In other words, it’s a mess.” ~ Mike Stone, viroLIEgy.com
“COVID-19” natural remedies : calendula, licorice, Chinese skullcap, rosemary, andrographis, artemisia, dandelion. Use the whole plant (root and leaf) of dandelion - make it very bitter. Melatonin, vitamin C, vitamin D, propolis tincture. Turpentine, garlic steam (see Jennifer Daniels, MD or Andrew Kaufman, MD) Support multiple detox pathways with Epsom Salt soaks, castor oil liver packs with heat, coffee enemas, saunas.
Midway through Martin gets real: the spike protein in the injections is a pathogen that creates auto-immunity. As I have been posting for months and months, there was/is no pathogenic 'virus' making people sick. The pandemic began with the injections. It is a pLandemic of extraoridinary dimension.
Forwarded from Dr David Martin
Media is too big
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How to remove self-generating spike protein toxins? How to know (test for) what my clients have had injected into them? I read that newly-incorporated bio-tech companies aligned with the likes of Scripps are moving to Florida to sell drugs for 'rare' diseases. IOW perhaps (likely) staged to profit from the i jury caused by these injections. And Martin says DeSantis won't meet with him. Ya don't say?!!
Nitric oxide, sulfates and pH ... at home, easy-to-use, inexpensive test strips essential for knowing your basic biochemistry.
MORE THEORIES ON HEALTH (from germs, terrain and on to t-helper cells and antibodies ... all theories!)
"The Th1/Th2 theory is one attempt at understanding immune regulation, and Th1 and Th2 cells are its key players. This theory dates back to studies on mouse immune cells in the 80s. However, it is still considered controversial and it’s not without limitations and discrepancies. More large-scale human studies are needed to determine its validity [1].
According to the Th1/Th2 theory [1]:
Th1 cells drive the so-called type-1 pathway (“cellular immunity”). They are thought to be involved in fighting viruses and other pathogens that enter cells, getting rid of cancerous cells, and triggering delayed-type hypersensitivity (DTH) skin reactions.
Th2 cells drive the type-2 pathway (“humoral immunity”). They are hypothesized to increase antibody production and fight invaders that are outside cells. They may be involved in tolerance of organ transplants (xenografts) and of the fetus during pregnancy."
"The Th1/Th2 theory is one attempt at understanding immune regulation, and Th1 and Th2 cells are its key players. This theory dates back to studies on mouse immune cells in the 80s. However, it is still considered controversial and it’s not without limitations and discrepancies. More large-scale human studies are needed to determine its validity [1].
According to the Th1/Th2 theory [1]:
Th1 cells drive the so-called type-1 pathway (“cellular immunity”). They are thought to be involved in fighting viruses and other pathogens that enter cells, getting rid of cancerous cells, and triggering delayed-type hypersensitivity (DTH) skin reactions.
Th2 cells drive the type-2 pathway (“humoral immunity”). They are hypothesized to increase antibody production and fight invaders that are outside cells. They may be involved in tolerance of organ transplants (xenografts) and of the fetus during pregnancy."