Heather Rae, Functional Health Practitioner
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health coach, specializing in alchemical biochemistry, genomics, anatomy, and bio-energetics
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Inflammation and Its Discontents: The Role of Cytokines in the Pathophysiology of Major Depression
"VIRUS" means poison, toxin or solvent. Psychopaths twisting words will have everyone believe that 'viruses' are alive like bacteria (they are not) and can be 'killed" (they cannot) and that our immune cells create antibodies (proteins) to unique microbiota (they do not).
ROMANS 1.18
For the wrath of God is revealed from heaven against all ungodliness and unrighteousness of men who suppress the truth in unrighteousness.
If you have inflammation in 'open spaces' (lungs, intestines, sinuses, vagina) as well as auto-immune (soft tissue inflammation) and dysbiosis (pathogen imbalances and leaky gut) ... in other words something akin to 'long haul covid' or 'mast cell activation and histamine' be careful with supplements because they can turn on more inflammation and backfire on you, if not taken carefully, in a logical sequence.
"Complex health issues like chronic fatigue, autoimmune disorders, and others always involve multiple possible factors–including body and brain chemistry, environment, health status, and genetics–that may vary from one person to another."
Fatty Acid Desaturase 1 (FADS1) Variants in FADS1 genes reduce the amount of anti-inflammatory resolvins, protectins and prostaglandins. Guess what blocks FADS1 genes? mRNA 'viruses'
you don't need to speak/read German to understand this graph ... it's a graph of side effects (including deaths) tracked by insurance companies. in short, the pandemic *is* the 'vaccine'
"Pfizer Vax Trial Manager Brook Jackson’s Shocking Revelations During Her First Sit Down w/ Paine and Former Blackrock’s Ed Dowd
Prepare for a rollercoaster ride here as we trek where few have gone before. I think perhaps this is the first interview of someone who was at Ground Zero of the scamdemic — the so called ‘vax’ trials. And it is as ugly as you can imagine."
COVID INJECTIONS & BLOOD PRESSURE MEDS ... 2 YEARS DOWN THE ROAD
Where it reads “COVID virus” insert “COVID injection” ... the spike protein of the un-isolated mythical SARSCov2 ‘virus’ (and the spike protein of the jabs) is said to enter the cell via the ACE2 enzyme in cells of multiple organs causing inflammation. What happens when someone is taking blood pressure meds that act on the same enzymes of the jab, takes the jab? Two years into the fraud of COVID, the numbers of CVD and deaths indicate what happens!

ACE inhibitor: lethal; ARB: life-giving.

“ACE inhbitors and angiotensin II receptor blockers [ARBs] are different blood pressure medication; receptors in the lung are the entry portal for COVID-19 virus; ACE inhibitors prohibit formation of angiotensin and the lungs express more receptors leading to the virus having more open doors; selenium is a natural ACE inhibitor and should be avoided; antiogtensin II receptor blockers (ARB) close the doors so that the virus cannot enter; Losartan is an example of an ARB that may be helpful.”
I can’t write about ‘viruses’ and ‘antibodies’ without feeling like Alice in Wonderland, or Dorothy on the Yellow Brick Road ... this road of virology and immunology as we know it today is a muck-filled trench of contradictions and non-sense. Here’s a sentence from a presentation in 2020 on Lyme that sounds very much like what we know about the computer-generated SARS ‘virus’. “The strain used for lab testing (B31) was a lab strain and never collected from a real human patient.”
ANTI-BODIES (think about that word for a minute) .... SCIENCE OF PROTEINS https://viroliegy.com/category/antibodies/
In Summary:
• Antibodies are said to provide potent protection against the flu and other “viruses”
• Vaccines are designed to stimulate antibody production
• Researchers measure antibodies to gauge the strength of the immune response to the infection
• However, questions remain about immune factors that drive the magnitude of the antibody response, including why flu infections do not always trigger a rise in blood antibodies
• The immune factors that explain why some people mount a strong antibody response and others don’t has not been well understood
• Airway monocytes often show up as correlates of severe disease, yet they were associated with protective immune responses in a recent study on influenza
• This was said to point to the “fact” that many immune factors play protective and pathogenic roles at the same time
“Taken all together, these studies provide indirect evidence that the unseen hypothetical/theoretical particles known as antibodies can be found in some cases of disease. These studies also provide evidence that these unseen hypothetical/theoretical particles are not found in many cases of disease. Antibodies may be associated with more severe disease but are also not present in cases of severe disease. They may be present in asymptomatic/mild cases of disease but are also not present in many asymptomatic/mild cases of disease.
The HAI gold standard of antibody titers > 40 has been correlated with “protection” in a few studies but this level has also been shown to be completely ineffective at providing “protection” in many studies post vaccination.
The problem for immunology and “antibodies” is that, just as in the case with virology and “viruses,” they rely primarily on correlation equaling causation. However, these correlations are inconsistent at best and many times non-existent and/or contradicted in future studies. Instead of admitting that they may be wrong by throwing out the old theories and starting over, the researchers try to cram new, contradictory findings/theories into the existing theory thus creating a nearly incomprehensible and overly confusing mosaic made up of pieces from completely unrelated puzzles.
This is why they can claim antibodies are “protective” even though they are not found in many cases of disease. This is how they can state that antibodies are a sign the immune system is working in regards to vaccinations yet they are also a sign one has a weakened immune system with HIV. This is why the presence of antibodies mean one has successfully fought off a disease and will be “protected” from future infections yet in the case of Dengue Fever, antibodies will make a reinfection worse and potentially fatal.
This is how we end up with multiple immune systems with factors that can be simultaneously protective and pathogenic. It is a long chain of confusing, confounding, and contradictory indirect evidence all rolled up into evolving incomprehensible theories. In other words, it’s a mess.” ~ Mike Stone, viroLIEgy.com